2026-10-10T11:49:39Z https://api.figshare.com/v2/oai

oai:figshare.com:article/34035920 2026-09-30T17:42:23Z category_13 category_14 category_46 category_734 category_106 category_122 portal_5 item_type_3 month_year_09_2026

<p>Mediation model output of ADHD and mixed pattern diseases as mediated by TNFR1A + B.</p> Sophie Breunig (25145387) Kristen M. Kelly (9340507) Lukas S. Schaffer (25145390) Jeremy M. Lawrence (25145393) Younga Heather Lee (25145396) Elizabeth W. Karlson (11935646) Andrew D. Grotzinger (6252026) Genetics Molecular Biology Immunology Biological Sciences not elsewhere classified Science Policy Mental Health literature extensively describes latent genomic factors highly attenuated associations g ., c g ., autoimmune crohn &# 8217 multiple disorder clusters examined immune markers 14 immune markers crp ], interleukin 13 psychiatric disorders immune disease link 6 mediated 60 multiple markers immune markers psychiatric disorders genetic link many immune immune diseases 11 immune xlink "> study explored significant mediators results indicated reactive protein psychiatric traits psychiatric relevance psychiatric relationships overlapping signal mediated diseases limited understanding investigated whether internalizing factor genetic links genetic liability diagnostic relevance biological precursor better explained <p>Mediation model output of ADHD and mixed pattern diseases as mediated by TNFR1A + B.</p> 2026-09-30T17:56:49Z Dataset Dataset 10.1371/journal.pgen.1012328.s018 https://figshare.com/articles/dataset/_p_Mediation_model_output_of_ADHD_and_mixed_pattern_diseases_as_mediated_by_TNFR1A_B_p_/34035920 CC BY 4.0